Welcome to Dinkum Prospect Healthcare, where your health is our highest priority. Our dedicated team of primary care professionals deliver comprehensive, evidence-based medical care designed to keep you well and address your concerns promptly.
Whether you need preventive screenings, chronic disease management, or expert guidance on your health goals, we stand ready to provide the trusted, individualized care you deserve.
You're more than just a medical chart. We take the time to listen, understand your concerns, and partner with you to create a care plan that supports your goals and lifestyle. Whether you're here for a routine checkup or managing a long-term condition, we're here to help.
We focus on preventive care, early diagnosis, and managing chronic conditions to keep you feeling your best.
Our primary care services include:
• Annual physical exams
• Preventive screenings
• Diagnosis and treatment of common illnesses
• Management of chronic conditions (e.g., diabetes, high blood pressure, asthma)
• Men's health
• Geriatric care
• Mental health support
• Nutrition and Weightloss
• Referrals to specialists when needed
A chronic wound fails to heal in a predictable timeframe, exposing patients to infections, pain, and diminished quality of life. Understanding the criteria that define a chronic wound empowers you to recognize when specialized care is needed and ensures timely intervention. In this article, you will discover the medical definition and timeframe that classify wounds as chronic, explore the main types and their distinguishing features, learn about common causes and risk factors, review diagnostic and assessment techniques, and examine evidence-based treatment and prevention strategies. Finally, you'll see how Dinkum Prospect Health's specialized wound care services in the DC Metro area deliver holistic support and expert interventions to help you heal.
A chronic wound is a non-healing wound that stalls in the repair process, typically remaining open beyond 4 to 12 weeks despite appropriate care. This stalled healing occurs because the normal phases—inflammation, proliferation, and remodeling—are disrupted by factors such as impaired blood flow or infection. For example, a diabetic foot ulcer that remains open after three months despite wound dressings and offloading demonstrates the hallmark of chronicity and signals the need for advanced interventions.
A chronic wound is defined as a wound that does not heal within the expected timeframe, typically beyond 4 to 12 weeks despite appropriate care. Wounds persisting beyond 12 weeks meet chronic criteria and require specialized wound care assessment.
This definition is crucial for understanding when specialized care is needed for a wound.
This stalled healing occurs because the normal phases—inflammation, proliferation, and remodeling—are disrupted by factors such as impaired blood flow or infection. For example, a diabetic foot ulcer that remains open after three months despite wound dressings and offloading demonstrates the hallmark of chronicity and signals the need for advanced interventions.
Medical definitions describe a chronic wound as a Medical Condition characterized by failure to progress through normal healing stages within an expected period. Physicians assess chronic wounds by evaluating tissue status, vascular supply, and underlying comorbidities to distinguish them from acute wounds, which should close within weeks under proper management.
Wound healing is expected to complete within four weeks in most cases and certainly by twelve weeks under optimal conditions.
Wounds persisting beyond four weeks are labeled delayed.
Wounds still unhealed at twelve weeks meet chronic criteria.
Persistent ulcers or non-closing surgical sites beyond this window necessitate specialized wound care assessment.
Chronic wounds develop when inflammatory cytokines remain elevated, proteases degrade vital extracellular matrix components, and growth factor signaling fails. Poor oxygenation and persistent infection maintain an inflammatory microenvironment that inhibits cell migration and collagen deposition. This biochemical imbalance prevents transition into proliferative healing and leads directly into necrotic tissue formation.
Healthcare providers look for clinical indicators that a wound is chronic:
Persistent pain disproportionate to size
Excessive exudate with malodor
Necrotic or sloughy tissue adhering to wound bed
Periwound erythema and edema suggesting ongoing inflammation
These symptoms often herald complications such as osteomyelitis, guiding clinicians to escalate care and imaging.
Chronic wounds fall into four primary categories based on etiology and patient risk profile:
Wound Type Underlying Cause Typical Duration
Diabetic Foot Ulcer Neuropathy & microvascular disease > 12 weeks despite care
Venous Leg Ulcer Venous insufficiency & hypertension Persistent venous stasis
Pressure Ulcer Prolonged pressure & shear forces Stages II–IV over 4+ weeks
Arterial Ulcer Peripheral arterial disease & ischemia Delayed capillary refill
Chronic wounds are categorized into several types based on their etiology, including diabetic foot ulcers, venous leg ulcers, pressure ulcers, and arterial ulcers. Each type has unique characteristics and requires targeted assessment and therapy.
This categorization helps in understanding the different causes and treatments for chronic wounds.
Each type exhibits unique pathophysiology and requires targeted assessment before therapy.
A diabetic foot ulcer develops when diabetic neuropathy reduces pain perception and peripheral artery disease impairs circulation to pressure-bearing areas of the foot. Risk factors include chronic hyperglycemia, foot deformities, and improper footwear. These ulcers often present as deep, punched-out lesions on weight-bearing zones and can progress rapidly without glycemic control and offloading interventions.
Diabetic foot ulcers develop due to diabetic neuropathy and peripheral artery disease. Risk factors include chronic hyperglycemia, foot deformities, and improper footwear, which can lead to deep lesions on weight-bearing zones.
Understanding these risk factors is essential for prevention and early intervention in diabetic patients.
Venous leg ulcers arise from chronic venous insufficiency, where dysfunctional valves lead to edema and tissue hypoxia. Diagnosis involves duplex ultrasound to assess reflux, measurement of ankle-brachial index to exclude arterial disease, and inspection for hemosiderin staining or lipodermatosclerosis. Compression assessment confirms that graduated stockings or bandaging can support improved venous return.
Venous leg ulcers arise from chronic venous insufficiency. Diagnosis involves duplex ultrasound to assess reflux, measurement of the ankle-brachial index to exclude arterial disease, and inspection for hemosiderin staining or lipodermatosclerosis.
This outlines the diagnostic steps for identifying venous leg ulcers.
Stage I: Intact skin with non-blanchable redness
Stage II: Partial-thickness skin loss
Stage III: Full-thickness skin loss through subcutaneous tissue
Stage IV: Extensive destruction into muscle or bone
Immobility, moisture, and nutritional deficits escalate risk, making repositioning protocols and support surfaces essential.
Pressure ulcers are staged based on tissue damage, ranging from Stage I (intact skin with non-blanchable redness) to Stage IV (extensive destruction into muscle or bone). Immobility, moisture, and nutritional deficits increase the risk.
This staging system helps in assessing the severity of pressure ulcers and guiding treatment.
Arterial ulcers reflect ischemia from peripheral artery disease, appearing as small, deep wounds on distal toes or shin. They present with cool, hairless surrounding skin and delayed capillary refill. Revascularization via angioplasty or bypass improves perfusion, while antiplatelet therapy, wound dressings, and pain management support healing once blood flow is restored.
Arterial ulcers result from ischemia due to peripheral artery disease, appearing as small, deep wounds on distal toes or shin. Revascularization improves perfusion, while antiplatelet therapy, wound dressings, and pain management support healing.
This describes the characteristics and treatment approaches for arterial ulcers.
Chronic wound formation stems from systemic and local contributors that disrupt healing.
Diabetes and Peripheral Artery Disease Diabetes impairs microvascular perfusion and leukocyte function, while PAD reduces macrovascular supply. Together, they stall repair and heighten infection risk.
Poor Circulation Venous hypertension and arterial insufficiency both limit oxygen and nutrient delivery, maintaining a hypoxic environment that degrades tissue and prolongs inflammation.
Immobility and Pressure Sustained pressure over bony prominences causes capillary collapse and tissue death. Shear forces further damage skin layers, compounding wound chronicity.
Other Health Conditions Obesity, smoking, malnutrition, and immunosuppression undermine host defense and slow collagen synthesis, raising the probability of non-healing wounds.
These overlapping factors create a complex milieu where multiple interventions are often required to restore healing progression.
Effective evaluation combines clinical observation with diagnostic tools to identify barriers to healing.
Effective evaluation combines clinical observation with diagnostic tools to identify barriers to healing. Physical examination, vascular assessments like the ankle-brachial index (ABI), imaging, and laboratory tests are used.
This highlights the methods used to diagnose and assess chronic wounds.
Physical examination assesses wound dimensions, appearance, and periwound tissue. Probing to bone checks for osteomyelitis, while sensory testing evaluates neuropathy. Inspection of pulse palpation at dorsalis pedis and posterior tibial arteries appraises arterial supply.
The ankle-brachial index (ABI) compares systolic pressures at the ankle and arm.
ABI ≥ 0.90 indicates adequate perfusion
ABI < 0.90 suggests arterial disease
ABI > 1.30 may reflect calcified vessels
ABI guides decisions on compression therapy in venous ulcers and revascularization for arterial wounds.
Radiographs identify osteomyelitis, while MRI delineates soft tissue involvement. Laboratory markers such as white blood cell count and C-reactive protein signal infection. Wound cultures guide targeted antibiotic therapy when signs of bacterial colonization emerge.
The TIME framework structures wound bed preparation:
Tissue: Debride nonviable tissue
Infection: Control microbial load
Moisture: Balance exudate with appropriate dressings
Edge: Promote cell migration at wound margins
Applying this protocol ensures each barrier to healing is systematically addressed before advanced interventions.
A multimodal approach accelerates closure and reduces complications, combining debridement, dressings, and adjunctive therapies.
A multimodal approach accelerates closure and reduces complications, combining debridement, dressings, and adjunctive therapies. Advanced options include hydrocolloids, alginate dressings, and foam dressings that manage exudate while preserving moisture.
This provides an overview of the treatment options available for chronic wounds.
Debridement removes necrotic tissue and biofilm, revealing a fresh wound bed and stimulating growth factor release. Techniques range from sharp surgical excision to autolytic methods using dressings that maintain a moist environment, each selected based on patient tolerance and wound characteristics.
Advanced options include hydrocolloids, alginate dressings, and foam dressings that manage exudate while preserving moisture. Negative pressure wound therapy (NPWT) applies suction to enhance perfusion and reduce edema. Hyperbaric oxygen therapy (HBOT) raises tissue oxygen levels to promote angiogenesis and collagen synthesis.
Dressing Type Characteristic Primary Benefit
Hydrocolloid Gel-forming matrix Maintains moist healing
Alginate Calcium-sodium fiber Absorbs heavy exudate
Foam Semi-permeable polyurethane Provides cushioning and moisture
NPWT Controlled negative pressure Improves perfusion and closure
Choosing the right dressing optimizes moisture balance and accelerates tissue granulation.
Graduated compression stockings or bandages reduce venous hypertension in leg ulcers, enhancing fluid return and minimizing edema. Offloading devices—such as total contact casts or specialized footwear—redistribute pressure away from diabetic foot ulcers, preventing further tissue damage and promoting closure.
Systemic antibiotics begin when clinical signs of infection—such as increased pain, erythema, or purulent discharge—appear in a chronic wound. Revascularization procedures restore arterial flow in ischemic ulcers, enabling dressings and debridement to be effective once adequate perfusion is reestablished.
Adopting a balanced diet rich in protein, vitamins, and minerals enhances tissue repair. Smoking cessation improves circulation, and maintaining healthy weight reduces pressure on vulnerable areas. Tight glycemic control in diabetes preserves microvascular integrity and optimizes white blood cell function.
Proactive lifestyle and self-care measures reduce the risk of new wounds and support existing wound closure. Adopting a balanced diet, smoking cessation, and maintaining a healthy weight are important.
This emphasizes the importance of lifestyle changes in preventing and supporting the healing of chronic wounds.
Regular skin inspection and moisturizing maintain barrier function, while repositioning every two hours relieves pressure. Specialized mattresses and cushions distribute forces evenly, preventing shear and friction that lead to pressure ulcers.
Daily foot examinations detect redness, blistering, or calluses early. Wearing well-fitting shoes and moisture-wicking socks prevents friction injuries. Prompt treatment of minor cuts with cleansing and antiseptic dressings forestalls progression into chronic ulcers.
If a wound shows no signs of epithelialization after four weeks or exhibits increased drainage, foul odor, or surrounding redness, it's essential to consult a wound care specialist. Early referral shortens healing time and prevents serious complications.
Our highly trained team employs advanced diagnostics and cutting-edge therapies to address complex wounds that have resisted standard care.
With a steadfast commitment to excellence, we deliver the focused, individualized treatment needed to promote healing, reduce complications and restore quality of life.
Our wound care services include sharp and enzymatic debridement, compression therapy fitting, and tailored dressing selection. Each treatment plan is developed by a multidisciplinary team led by experienced clinicians to address your unique needs.
By integrating primary care management of chronic conditions, nutritional guidance, and mental health support, our holistic model tackles underlying risk factors and enhances adherence. Addressing both physical barriers and emotional well-being creates a healing environment that fosters long-term recovery.
To arrange a consultation with our wound care specialists, call our DC Metro clinic or visit our online booking portal. Our telephone number is 240-559-4838 and our website address is www.dinkumprospecthealth.com to schedule an appointment.
Our team will guide you through insurance verification and schedule a timely evaluation to begin personalized wound management without delay.
A chronic wound that meets defined criteria requires prompt, specialized intervention to prevent complications. Recognizing prolonged healing, biochemical imbalances, and characteristic signs ensures you and your healthcare provider act swiftly. Dinkum Prospect Health's integrated wound care services deliver advanced treatments and holistic support to guide your healing journey. Reach out today to secure expert care and start closing the gap on chronic wounds.
Dinkum Prospect Healthcare is a primary, wound and mental healthcare practice. We are here to help you get back to living your very best self.
Dr. Motunrayo Adegorusi has years of medical experience. She has provided care for patients from all works of life. She is a compassionate and caring health care provider.
© 2025 All rights reserved. Privacy Policy